| Expression System | CHO |
| Aggregation | < 5% as determined by SEC-HPLC |
| Purity | > 95% as determined by SDS-PAGE |
| Endotoxin | < 1 EU/mg, determined by LAL gel clotting assay |
| Sterility | 0.2 μm Filtered |
| Target | NPR1 |
| Clone | XX-16 |
| Alternative Names | ANPRA, ANPa, GUC2A, GUCY2A, NPRA |
| Source/Isotype | Monoclonal Human IgG1(REEM), Kappa |
| Application | Flow Cytometry |
| Description | NPR1 (Non-expressor of Pathogenesis-Related Genes 1) is a key gene in the plant defense mechanism, primarily responsible for regulating Systemic Acquired Resistance (SAR), a global defense strategy activated in response to pathogen infection. In this process, NPR1 serves as a central regulatory factor, sensing and transmitting defense signals to activate downstream defense genes. Additionally, NPR1 plays a broad role in enabling plants to respond to various environmental stresses, contributing significantly to plant survival and adaptability. |
| Formulation | phosphate-buffered solution, pH 7.2-7.4. |
| Expression System | CHO |
| Aggregation | < 5% as determined by SEC-HPLC |
| Purity | > 95% as determined by SDS-PAGE |
| Endotoxin | < 1 EU/mg, determined by LAL gel clotting assay |
| Sterility | 0.2 μm Filtered |
| Target | NPR1 |
| Clone | XX-16 |
| Alternative Names | ANPRA, ANPa, GUC2A, GUCY2A, NPRA |
| Source/Isotype | Monoclonal Human IgG1(REEM), Kappa |
| Application | Flow Cytometry |
| Description | NPR1 (Non-expressor of Pathogenesis-Related Genes 1) is a key gene in the plant defense mechanism, primarily responsible for regulating Systemic Acquired Resistance (SAR), a global defense strategy activated in response to pathogen infection. In this process, NPR1 serves as a central regulatory factor, sensing and transmitting defense signals to activate downstream defense genes. Additionally, NPR1 plays a broad role in enabling plants to respond to various environmental stresses, contributing significantly to plant survival and adaptability. |
| Formulation | phosphate-buffered solution, pH 7.2-7.4. |
